Inside a real Revi report
The pages below are taken from the founder's own Revi Longevity report, with identifying details partially redacted.
Revi is built to turn a broad biomarker panel into a clear, prioritised plan — showing what matters most, why it matters, and what to do first.
Each report includes:
- a concise summary of your overall longevity and performance position
- the markers and systems that look strongest
- the areas with the greatest room for optimisation
- scores and rankings across 8 longevity systems
- your highest-impact actions based on your marker pattern
- supplement considerations only where your results support them
- deep dives into your 3 lowest-scoring systems
- additional optimisation opportunities flagged by your biomarkers
- your raw biomarker scores in the appendix
Rather than chasing every small deviation, Revi pinpoints the few levers most likely to improve your long-term health and performance.
This example is for illustration only. Revi reports do not provide medical diagnosis, and recommendations are personalised to the marker pattern, context, and results shown.
What will move the needle most
These are the highest-impact changes across your full profile — start with the core actions, then layer in the next most important changes, including targeted supplements. This sequence drives the biggest gains in drive, strength, and body composition — where your profile has the most to gain.
Your priorities & actions
Your HbA1c is 40.77 mmol/mol — in the upper half of the normal range (reference <42), worth optimising before it drifts toward the UK high-risk range of 42–47 mmol/mol. Your ApoB is 76 mg/dL — well under the reference ceiling, but above the longevity-optimal target of <60. Together these point to the same upstream lever: cholesterol clearance and metabolic flexibility.
Aim for 30–40 g total fibre daily, with at least 10 g from viscous sources. Practical recipe: 80 g oats at breakfast, 150 g cooked lentils or black beans at lunch, one tablespoon of psyllium husk stirred into water each evening — build up over two weeks to avoid digestive discomfort. Alongside this, keep saturated fat to under ~7% of daily energy intake — swap butter for olive oil, choose leaner meats, limit full-fat dairy.
Longevity Optimising HbA1c and ApoB are two of the strongest single levers for reducing long-term cardiovascular and metabolic disease risk.
Performance More stable post-meal glucose means more consistent energy across training days and fewer afternoon energy dips.
Day to day The sharp energy dips between meals may become less frequent within two to three weeks.
MetabolicLipid & HeartHbA1cApoBLDL CholesterolAdiponectinHDL Cholesterol
Your fasted Transferrin Saturation is 44.7%, which is high-normal and close enough to common investigation thresholds to justify a clean morning repeat with ferritin before making any iron-related changes. Combined with the wider red-cell context, the right direction here is to rule out iron overload first, not to add iron. The MCHC signal alone does not justify iron loading when TSAT is at this level.
Repeat fasted morning iron studies — Iron, TIBC, Transferrin Saturation, and Ferritin — in 6–8 weeks. If TSAT remains elevated or ferritin rises, discuss HFE genotype testing with your GP. Hold all iron-loading dietary changes and iron-containing supplements, including any multivitamin with iron, until iron status is clarified.
Longevity Catching hereditary haemochromatosis early is one of the highest-yield genetic screens available — untreated iron overload damages liver, pancreas, and heart over decades.
Performance If iron is genuinely tight at the cell-production level, the correct intervention only becomes clear once overload is ruled out.
Day to day A clean retest under fasted morning conditions gives a definitive baseline rather than chasing the MCHC signal in isolation.
Cellular EnergyTransferrin SaturationFerritinIronMean Cell Haemoglobin Concentration (MCHC)
Your vitamin D is 169 nmol/L — above the optimal band of 100–150, and well above sufficient (>50). Above ~125 nmol/L, additional supplementation provides no measurable benefit, so the goal is simply to bring you back into the evidenced optimal range.
Reduce your vitamin D supplement to a maintenance dose of around 800–1,000 IU/day — or pause it entirely for 8 weeks and reassess. Continue your vitamin K2 as normal; it's the D3 dose that needs to come down. Retest 25(OH)D at your next panel and resume only at the lower maintenance dose if you drop below 100 nmol/L.
Longevity Bringing vitamin D back into the optimal band of 100–150 nmol/L removes any theoretical long-term risk from chronic over-supplementation while keeping the full benefit of being above sufficient.
Performance There's no functional performance loss from this change. Above 125 nmol/L, additional vitamin D doesn’t measurably improve strength, recovery, or output.
Day to day You won’t feel different from this change. The lever is removing future risk, not creating current benefit.
NutrientVitamin D
Your HbA1c reflects average glucose over three months and is in the upper half of normal — worth understanding before it drifts further — but fasting glucose and HbA1c averages cannot show which specific meals or habits are driving the pattern — a continuous glucose monitor fills that gap directly.
Consider a 14-day glucose-sensor trial using a UK-available consumer option such as Abbott Lingo, or a self-funded pharmacy CGM option where appropriate. Use it to identify meal patterns, not to diagnose a condition. This is for pattern discovery, not diagnosis.
Longevity Identifying and correcting the specific dietary triggers behind a rising HbA1c is one of the most targeted long-term metabolic investments available.
Performance Understanding your personal glucose response to meals allows you to time nutrition around training for better energy and recovery.
Day to day Knowing exactly which foods spike your glucose removes the guesswork and makes the dietary changes feel concrete and manageable.
MetabolicHbA1cGlucose
Consistent sleep timing is a strong recovery and glucose-regulation lever, and it also creates cleaner conditions for interpreting the hormone retest. Use it to support training adaptation, energy stability, and retest quality — not as a direct hormone-correction strategy.
Set a fixed sleep window of 22:30–06:30 every night, including weekends, with room temperature at 17–19°C and no screens after 21:30. Keep wake time fixed even if sleep onset is delayed — consistency of timing matters more than total hours in the first two weeks.
Longevity Consistent sleep architecture supports hormonal regulation, immune resilience, and long-term cardiovascular health.
Performance Fixed sleep timing improves recovery quality, HRV, and the hormonal environment that drives training adaptation.
Day to day Motivation and drive may feel more consistent within two to three weeks of holding the window.
HormonesMetabolicEstradiolSHBGFree TestosteroneHbA1cFSH (Follicle Stimulating Hormone)
Not shown here, included in the full report:
Secondary priorities (additional patterns worth monitoring) and a 12-week "what to expect" projection.
Supplement strategy
Your supplement recommendations are built directly from your biomarker results and priority systems — not from a generic protocol. Each is graded as a core action (well-evidenced and well-matched to your markers) or a conditional consideration (worth weighing against current intake and retest priorities before starting).
Top strategy
Start with the highest-confidence, lowest-risk lever first. In this profile, that means fibre/psyllium as part of the cardiometabolic plan. Magnesium should be reviewed against current form, dose, and intake. Selenium should be considered only if dietary intake, thyroid context, and retest priorities support it.
Audit your existing stack
Pause or reduce your Vitamin D3 — your 25(OH)D reads 169 nmol/L, above the longevity-optimal band of 100–150. Drop to a maintenance dose of around 800–1,000 IU/day, or pause for 8 weeks and retest. Continue K2 as normal.
Retest in 12 weeks
At 12 weeks, retest Magnesium, Anti-Thyroid Peroxidase Antibody, AA:EPA Ratio, and LDL Cholesterol — the markers tied to your supplement protocol — to confirm the stack is working.
Supplement considerations shown
2 core · 1 conditional
Selected because: Optimisation Opportunity — Magnesium 53% toward the reference floor
Why it is here
You mentioned taking magnesium in your declared supplements — worth confirming your current product is magnesium glycinate or threonate (the better-absorbed forms) at a meaningful dose, as your Magnesium sits 53% below Revi Optimal, suggesting the current approach may not be fully resolving the gap.
How it could help
Magnesium glycinate or threonate could help support insulin sensitivity, sleep quality, and cortisol regulation — all relevant given your metabolic and stress picture.
Markers & systems
MagnesiumHbA1cCortisolStress & RecoveryMetabolic
Why this ranked here
Your Magnesium is trending low at 53% below Revi Optimal despite declared supplementation, making form and dose confirmation the immediate priority before adding anything else.
Selected because: Optimisation Opportunity — Anti-Thyroid Peroxidase Antibody 54% toward the reference ceiling
Why it is here
Anti-TPO is within the lab reference range but above Revi's optimal zone. Selenium may be considered as a conditional thyroid-support option after checking current dietary intake, existing supplement use, and retest priorities. It is not a core first-line action from this panel.
How it could help
Selenium may support thyroid peroxidase enzyme function and could modulate antibody load over a 12–16 week period when there is a clear clinical rationale. Anti-TPO sits within the lab reference range (<34), so this is preventative consideration only, not therapeutic intervention.
Markers & systems
Anti-Thyroid Peroxidase AntibodyThyroid Stimulating Hormone (TSH)Free Thyroxine (FT4)
Why this ranked here
Considered conditionally because anti-TPO sits above Revi's optimal zone but within the lab reference range. Worth reviewing dietary selenium intake, existing supplement overlap, and retest priorities before starting.
Selected because: Optimisation Opportunity — HbA1c 82% toward the reference ceiling • LDL Cholesterol 74% toward the reference ceiling • Adiponectin 66% toward the reference floor (+1 more)
Why it is here
Your panel shows four converging signals that psyllium is well matched to: LDL Cholesterol 74% above Revi Optimal, HbA1c 82% above Revi Optimal, Adiponectin 66% below Revi Optimal, and Homocysteine trending mildly high — psyllium's soluble fibre mechanism is one of the broadest low-risk levers across this pattern.
How it could help
Psyllium husk could help support LDL reduction via bile acid sequestration, improve post-meal glucose handling, and contribute to the fibre foundation that supports adiponectin and metabolic resilience over time.
Markers & systems
LDL CholesterolHbA1cAdiponectinHomocysteineLipid & HeartMetabolic
When to add
Add from the outset alongside Tier 1 — one tablespoon (approximately 5–7g) in a large glass of water each evening, building to twice daily if tolerated.
Before starting: these recommendations identify which compounds the evidence supports for your profile. Specific doses, brands, forms, and timing are best agreed with a qualified practitioner — your GP, pharmacist, or a registered nutritional therapist — before you begin.
Where You Stand
This system has a big say in your energy stability, body composition, long-term health, and how consistently you perform across the day. Several markers here are reassuring — your triglycerides are optimal, your waist-to-height ratio is lean, and your blood pressure is excellent. The main signal is an HbA1c reading of 40.77 mmol/mol sitting still below the UK high-risk range of 42–47, but close enough to make glucose stability a sensible optimisation focus, alongside a low adiponectin and below-reference HDL, which together are consistent with a glucose-handling and cardiometabolic-resilience pattern worth addressing now.
Metabolic
OptimalStrongOpportunityFocus
Your personalised longevity score, from 8 scored biomarkers
Biomarker distribution profile8 biomarkers
4 Optimal Zone2 Opportunity: In Range2 Focus: Outside Range
Anchor Biomarkers Critical to system performance
Primary anchor
Insulin pmol/L
Secondary anchor
HbA1c mmol/mol
Supporting Biomarkers Contribute to system performance
Glucose mmol/L
Outside RangeHDL Cholesterol mmol/L
Outside RangeAdiponectin µg/ml
In RangeTriglycerides mmol/L
OptimalC-Peptide ng/ml
Beyond Optimal
Fix this first
Primary Driver
Low protective cholesterol and a near-ceiling HbA1c, consistent with a glucose-regulation pattern under pressure — partly shaped by an extended fast at draw time
What this means
Your body's capacity to buffer cardiovascular and metabolic stress is lower than it should be at 36. Left unaddressed, this pattern may gradually increase long-term cardiometabolic load, so it is worth addressing while the rest of your profile is strong.
Why this is happening
The extended 17-hour fast explains the low glucose and insulin readings — those are expected, not alarming. What holds regardless of draw state is the near-threshold HbA1c and suppressed protective cholesterol, suggesting the glucose-regulation system is working harder than it should over months, not just this morning.
If you notice this
Energy that dips sharply between meals, or recovery feeling slower than your training volume would justify.
Start here
Add 30–40 g of dietary fibre daily for 8 weeks — start with 80 g oats at breakfast, 150 g lentils at lunch, and one tablespoon psyllium husk each evening — then retest with a standard 10–12 hour fast.
Your highest-leverage actions
Add 30–40 g total dietary fibre daily, with at least 10 g from viscous sources: 80 g oats at breakfast, 150 g cooked lentils or black beans at lunch, and one tablespoon psyllium husk stirred into water each evening. Build up over two weeks.
Viscous fibre slows glucose absorption after meals and feeds gut bacteria that signal the body to produce more adiponectin — the hormone that keeps cells responsive to insulin — making this the most direct dietary lever for both markers. The payoff you feel: steadier energy across the day, fewer afternoon slumps, and better body composition over time.
Links to: HbA1c Adiponectin
First shift in post-meal glucose stability at 4–6 weeks; HbA1c and adiponectin changes measurable at 12 weeks.
Add two resistance training sessions per week to your existing training load, built around compound lifts — squats, deadlifts, rows, and overhead press — at 70–80% of your one-rep max, 6–10 working sets per muscle group per week, with progressive overload applied weekly.
Resistance training builds muscle mass, which acts as the body's primary glucose-storage organ — more muscle means the body handles blood sugar more cleanly, may help move HbA1c toward optimal and raise adiponectin over time, with secondary benefit to the heart-protective cholesterol marker. What this does for you: stronger performance in training, better body composition, and more consistent energy across the day.
Links to: HbA1c Adiponectin HDL Cholesterol
Glucose handling improvements begin at 6–8 weeks; HbA1c and adiponectin shifts measurable at 12–16 weeks.
Shift your eating window to a consistent 8-hour window aligned to daylight — for example 10:00–18:00 — on at least 5 days per week. Plain water, black coffee, and black tea are permitted outside the window. No calorie counting required within the window.
Aligning your eating window to daylight gives the liver and pancreas an extended daily rest period, improving how efficiently the body handles glucose independently of any weight change — the mechanism is circadian alignment of insulin signalling, not caloric restriction. The payoff you feel: steadier afternoons, more stable energy between meals, and improved metabolic flexibility over time.
Links to: HbA1c Adiponectin Glucose
Early glucose stability improvements at 4–6 weeks; HbA1c shift measurable at 12 weeks. Note: your Glucose reading of 3.91 mmol/L was drawn after a 17.2-hour fast — the low reading is the expected physiological response to extended fasting, not dysfunction. Retest after a standard 10–12 hour fast to confirm true fasting baseline before treating Glucose as a primary concern.
Advanced protocols for this system
Beginner: 30–90 sec cold shower at end of regular shower, daily. Standard: 10–15°C water, 2–5 min, 3–4 sessions/week (post-training or morning). Advanced: 3–8°C, 2–3 min, 2–3 sessions/week. Do not exceed 11 minutes total weekly exposure.
Activates brown fat — the calorie-burning fat that also improves insulin sensitivity — and triggers a controlled noradrenaline surge that builds stress resilience. Also conditions the vagus nerve, supporting recovery and mood regulation.
Your HbA1c sits at 40.77 mmol/mol — still below the UK high-risk range of 42–47, but close enough to make glucose stability a sensible optimisation focus — and cold exposure may provide a small additional glucose-handling stimulus, but it is secondary to fibre, fat quality, resistance training, and glucose-pattern awareness. What you may notice: more stable energy across the day and improved recovery between training sessions.
Links to: HbA1c
80–100°C, 15–30 min per session, 3–4 sessions per week. Finish with a cool shower or rest, rehydrate with 500 ml water plus a pinch of salt.
Produces a cardiovascular load similar to moderate exercise — heart rate rises, blood vessels widen, and the body activates heat shock proteins that support cellular repair. Long-term users show meaningful cardiovascular and all-cause mortality benefits.
Your HDL Cholesterol sits 24.5% below the reference range, and given your resting pulse of 50 BPM indicates aerobic fitness is already well-developed, sauna provides a meaningful cardiovascular stimulus that supports the heart-protective cholesterol marker through a different pathway than aerobic training. What you may notice: improved cardiovascular resilience and recovery between hard training days.
Links to: HDL Cholesterol
Diagnostics & Measurement
Use a UK-available consumer glucose-sensor option such as Abbott Lingo, or a self-funded pharmacy CGM option where appropriate. Wear it for 14 days while eating normally. Use the data for pattern discovery, not diagnosis.
A fasting glucose and HbA1c report an average — they miss the post-meal peaks that drive the trajectory. A CGM converts abstract numbers into concrete decisions: which breakfasts spike you, how training timing changes the curve, whether afternoon coffee nudges glucose up. Especially valuable when HbA1c sits near the pre-diabetes screening threshold.
Your HbA1c at 40.77 mmol/mol is still below the UK high-risk range of 42–47, but close enough to make glucose stability a sensible optimisation focus — a 14-day CGM trial would reveal exactly which meals or habits are driving the trajectory, turning an abstract number into a precise, actionable target before the next retest. This is especially valuable given your Glucose was drawn after a 17.2-hour fast, making the single fasting reading uninterpretable as a true baseline.
Links to: HbA1c Glucose